MY BATTLE AGAINST CANCER: Survivor protocol : foreword by Thomas Seyfried

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Muri, E.M.F.; Nieto, M.J.; Sindelarand, R.D.; Williamson, J.S. Hydroxamic Acids as Pharmacological Agents. Curr. Med. Chem. 2002, 9, 1631–1653. [ Google Scholar] [ CrossRef] kimster, I have liked at least on a conceptual level this article https://arxiv.org/pdf/1407.7622.pdf One anomaly I cannot understand though is that his PSA has been at below 0.1 for over a year now and yet he still has a prostate. How is this possible?

Lee, I.-K.; Han, M.-S.; Lee, M.-S.; Kinm, Y.-S.; Yun, B.-S. Styrylpyrones from the medicinal fungus Phellinusbaumii and their antioxidant properties. Bioorg. Med. Chem. Lett. 2010, 20, 5459–5461. [ Google Scholar] [ CrossRef] [ PubMed] We have seen that saturated fatty acids of 16 carbons, accompanied by high-fat diets, stimulate AMP deaminase. The resultant decrease in AMP inhibits AMP kinase, canceling its inhibitory action over ACC, which is activated. This boosts the fatty acid synthesis pathway, while malonyl-CoA turns off the fatty acid degradation into acetyl-CoA. If DAG decreases (and converts to TAG) it will not stimulate PKC. Thus, the CPI 17 inhibitor of PP1 does not form, and the phosphatase dephosphorylates and activates PK and PDH, opening the glycolytic supply of acetyl-CoA. On the contrary, if DAG increases (following the action of Growth hormone, for example) PKC is stimulated, forming the CPI 17 inhibitor of PP1, which maintains the phosphorylation of PK and PDH. Since both the fatty acid and glycolytic acetyl-CoA supplies are closed, tumor cells must then use ketone bodies from the liver to create their acetyl-CoA. Pournourmohammadi, S.; Grimaldi, M.; Stridh, M.H.; Lavallard, V.; Waagepetersen, H.S.; Wollheim, C.B.; Maechler, P. Epigallocatechin-3–gallate (EGC) activates AMPK through the inhibition of glutamatedehydrogenase in muscle and pancreatic Bcells: A potential beneficial effect in the pre-diabetic state. Int. J. Biochem. Cell Biol. 2017, 88, 220–225. [ Google Scholar] [ CrossRef][ Green Version]This is exciting! I was wondering whether reports of cancer patients trying prolonged water only fasts might be in the literature. Apparently, recently a report of a 21 day water only fast was reported for a cancer patient which resulted in a long term remission (see the two articles below). There are some caveats here: The type of cancer involved stage IIIa, low-grade follicular lymphoma is highly treatable with current therapies, so one wonders to what extent it is ethical to not first give standard of care. Also this type of cancer appears to have prolonged survival so it is not obvious how effective the diet intervention was. He’s had no traditional treatment, but simply did lots of fasting and taking supplements and he’s been diagnosed as cancer free since last June. His metastasis are gone and only the old scars remain on his bone scans. In general, HDACs, together with other compounds (NO donors), control the embryonic to adult transition for many protein isoforms. This depends, at the epigenetic level, on metabolites formed in glycolytic-ketogenic-ammonotellic fetal metabolism, or metabolites formed in adult oxidative metabolism. In mammals, a series of proteins of the “aquatic fetus” thus adapt to life in an environment with air and gravity [ 9].

Nelson, D.C.; Riseborough, J.A.; Flematti, G.R.; Stevens, J.; Ghisalberti, E.L.; Dixon, K.W.; Smith, S.M. Karrikins discovered in smoke trigger Arabidopsis seed germination by a mechanism requiring GibberellicAcid synthesis and light. Plant Physiol. 2009, 149, 863–873. [ Google Scholar] [ CrossRef][ Green Version]

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But having Psa at 0.1 does not mean he has remission. After 4 doses of Lu177 my Psa went from 25 before Lu177 to 0.32 at 12 months later in Nov 2019r, and doctors were amazed, but I have always known that to be due to Xtandi working, because I had begun it in April 2019. Konishi, H.; Fujiya, M.; Tanaka, H.; Ueno, N.; Moriichi, K.; Ikuta, K.; Akutsu, H.; Tanabe, H.; Kohgo, Y. Probiotic-derived ferrichrome Inhibits colon cancer progression via JNK-mediated apoptosis. Nat. Commun. 2016, 7, 12365. [ Google Scholar] [ CrossRef]

This book tells my story, the story of a carefree man leaving a happy life in Miami, who suddenly falls into the nightmare of the disease whose just name scares cancer. Mine is incurable, or rather all of mine because the prostate, bones and lymph nodes are affected. Despite the terrible prognosis from my oncologists, I have decided that I will not die of cancer. Do you think that's impossible? Yet I should be dead by the time of this writing. The point is, I have never felt this good in years and my latest results point to remission!

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There are dozens (hundreds maybe) of debunking videos on YouTube about him from actual qualified medical practitioners.